foam cells การใช้

• Foam cells eventually die and further propagate the inflammatory process.
• Accumulation of lipid droplets induce the modification of macrophages to foam cells.
• ANGPTL4 suppresses foam cell formation to reduce atherosclerosis development.
• Macrophages accumulate modified lipid particles and become foam cells.
• Low-density lipoprotein ( LDL ) cholesterol is contained by a foam cell.
• Instead CXCL5 has a protective role in atherosclerosis by directly controlling macrophage foam cell formation.
• Foam cells form the fatty streaks of the plaques of atheroma in the tunica intima of arteries.
• Lysis of foam cells resulted in Atherosclerotic plaques and such plaques rupture and blocked the thrombotic vessel.
• These LDL molecules are oxidized and taken up by macrophages, which become engorged and form foam cells.
• These foam cells often become trapped in the walls of blood vessels and contribute to atherosclerotic plaque formation.
• The oxidized LDL accumulates in the macrophages and other phagocytes, which are then known as foam cells.
• The formation of foam cells and their continued accumulation in the intima lead to the formation of fatty streaks.
• After they accumulate large amounts of cytoplasmic membranes ( with associated high cholesterol content ) they are called foam cells.
• Scavenger receptors typically are found on macrophages, with cholesterol laden macrophages being better known as " foam cells ".
• Foam cells are the body's way of trying to get rid of bad cholesterol from the blood vessels.
• The immune system's specialized white blood cells ( macrophages ) absorb the oxidized LDL, forming specialized foam cells.
• Foam cells are formed when the body sends macrophages to the location of a fatty deposit on the blood vessel walls.
• Foam cells do not give off any explicit signs or symptoms, but they are part of the origin of atherosclerosis.
• Atherosclerosis is caused by the accumulation of macrophages containing cholesterol ( foam cells ) in artery walls ( in the intima ).
• In Germany he became the first to describe the cholesterinesterphagozyten, which derive from macrophages and today are known as foam cells.